work: Basic engineering models have been developed to describe cerebro- cerebellar interaction in the control of single joint and two-joint limb stabilization and movement [3, 2]. These models adhere to known neu-roanatomy and spinomuscular physiology and propose quantitative descriptions

originate from distinct output channels within the cerebellar nuclei. The cerebral cortical area that is the main target of each output channel is a major source of input to the channel. Thus, a closed-loop circuit represents the major architectural unit of cerebro-cerebellar interactions. The outputs

: Facilitated conditioning suggests that an enhanced excitability in the cerebellum occurs as part of a disrupted CCTCC in schizophrenia. The enhanced cere-bellar-mediated associative learning may be maladaptive in the context of normal cerebro–cerebellar interactions, leading to the characteristic motor

, if indirect, method for mapping brain circuits in humans. In the first part of this dissertation I use fcMRI to map cerebro-cerebellar networks (Paper I). These networks replicate known anatomical circuits from non-human primates. Other regions in the

Although cognitive impairment after cerebellar damage has been widely reported, the mechanisms of cerebrocerebellar interactions are still a matter of debate. The cerebellum is involved in sequence detection and production in both motor and sensory domains, and sequencing has been proposed