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Functional discovery via a compendium of expression profiles. Cell 102:109
, 2000
"... have been devised to survey gene functions en masse either computationally (Marcotte et al., 1999) or experimentally; among these, highly parallel assays of ..."
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Cited by 537 (8 self)
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have been devised to survey gene functions en masse either computationally (Marcotte et al., 1999) or experimentally; among these, highly parallel assays of
The Pathophysiology of Mitochondrial Cell Death This copy is for your personal, non-commercial use only. clicking here.colleagues, clients, or customers by , you can order high-quality copies for yourIf you wish to distribute this article to others here.f
- JULY 2004 629 DOI: 10.1126/science.1099320 , 626 (2004);305 Science
"... In the mitochondrial pathway of apoptosis, caspase activation is closely linked to mitochondrial outer membrane permeabilization (MOMP). Numerous pro-apoptotic signal-transducing molecules and pathological stimuli converge on mitochondria to induce MOMP. The local regulation and execution of MOMP in ..."
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Cited by 323 (2 self)
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In the mitochondrial pathway of apoptosis, caspase activation is closely linked to mitochondrial outer membrane permeabilization (MOMP). Numerous pro-apoptotic signal-transducing molecules and pathological stimuli converge on mitochondria to induce MOMP. The local regulation and execution of MOMP
adipose tissue: function and physiological significance. Physiol Rev 84
, 2004
"... A. Norepinephrine signaling through �3-receptors leads to thermogenesis 280 B. Thermogenesis is due to activation of UCP1 through lipolysis 283 C. The �2-adrenergic pathway inhibits thermogenesis 288 D. The �1-adrenergic pathway and the cell membrane events 289 III. The Life of the Brown Adipocyte I ..."
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Cited by 308 (4 self)
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A. Norepinephrine signaling through �3-receptors leads to thermogenesis 280 B. Thermogenesis is due to activation of UCP1 through lipolysis 283 C. The �2-adrenergic pathway inhibits thermogenesis 288 D. The �1-adrenergic pathway and the cell membrane events 289 III. The Life of the Brown Adipocyte
How do glucocorticoids influence stress responses? Integrating permissive, suppressive, stimulatory and preparative actions. Endocrine Rev 21:55–89
, 2000
"... The secretion of glucocorticoids (GCs) is a classic endocrine re-sponse to stress. Despite that, it remains controversial as to what purpose GCs serve at such times. One view, stretching back to the time of Hans Selye, posits that GCs help mediate the ongoing or pending stress response, either via b ..."
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Cited by 302 (6 self)
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basal levels of GCs permitting other facets of the stress response to emerge efficaciously, and/or by stress levels of GCs actively stimulating the stress response. In con-trast, a revisionist viewpoint posits that GCs suppress the stress response, preventing it from being pathologically overactivated
Socioeconomic Status and Health: The Challenge of the Gradient
- SOCIAL INFLUENCES ON BIOLOGY AND HEALTH A. BASIC PROCESSES 71
"... ..."
Efficient inference of cancer progression models
, 2014
"... A tumor is thought to result from successive accumulation of genetic alterations – each resulting population manifesting itself with a novel ‘cancer phenotype. ’ In each such popu-lation, clones of higher fitness, contributing to the selection of the cancer phenotype, enjoy a Darwinian selective adv ..."
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A tumor is thought to result from successive accumulation of genetic alterations – each resulting population manifesting itself with a novel ‘cancer phenotype. ’ In each such popu-lation, clones of higher fitness, contributing to the selection of the cancer phenotype, enjoy a Darwinian selective
Nitric Oxide and Peroxynitrite in Health and Disease
"... II. Nitric Oxide 317 A. Discovery of nitric oxide as a biological molecule 317 B. The selective reactivity of nitric oxide 319 C. Diffusion and signaling properties of nitric oxide 319 ..."
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Cited by 263 (5 self)
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II. Nitric Oxide 317 A. Discovery of nitric oxide as a biological molecule 317 B. The selective reactivity of nitric oxide 319 C. Diffusion and signaling properties of nitric oxide 319
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