Abstract not found

Numerous studies show that happy individuals are successful across multiple life domains, including marriage, friendship, income, work performance, and health. The authors suggest a conceptual model to account for these findings, arguing that the happiness–success link exists not only because success makes people happy, but also because positive affect engenders success. Three classes of evidence—crosssectional, longitudinal, and experimental—are documented to test their model. Relevant studies are described and their effect sizes combined meta-analytically. The results reveal that happiness is associated with and precedes numerous successful outcomes, as well as behaviors paralleling success. Furthermore, the evidence suggests that positive affect—the hallmark of well-being—may be the cause of many of the desirable characteristics, resources, and successes correlated with happiness. Limitations, empirical issues, and important future research questions are discussed.

The online version of this article, along with updated information and services, is located on the

Evidence of the health-promoting influence of primary care has been accumu-lating ever since researchers have been able to distinguish primary care from other aspects of the health services delivery system. This evidence shows that primary care helps prevent illness and death, regardless of whether

adipocytes 294 IV. How Significant Is Brown Adipose Tissue? 295 A. Parameters of activation and recruitment 295 B. How to establish brown adipose tissue involvement 297 V. Thermoregulatory Thermogenesis 298 A. In acute cold, thermogenesis results from shivering 298 B. Classical nonshivering thermogenesis

from 6 studies conducted in 4 countries. Substantial similarity in means, standard deviations, and correlations among the scales, and in correlations between scales and demographic variables, is found for both men and women in all studies. Reliability is good for most scales. Results suggest

Using a process model of emotion, a distinction between antecedent-focused and response-focused emotion regulation is proposed. To test this distinction, 120 participants were shown a disgusting film while their experiential, behavioral, and physiological responses were recorded. Participants were told to either (a) think about he film in such a way that they would feel nothing (reappraisal, a form of antecedent-focused emotion regulation), (b) behave in such a way that someone watching them would not know they were feeling anything (suppression, a form of response-focused motion regulation), or (c) watch the film (a control condition). Compared with the control condition, both reappraisal and suppression were effective in reducing emotion-expressive behavior. However, reap-praisal decreased isgust experience, whereas uppression increased sympathetic activation. These results suggest that these 2 emotion regulatory processes may have different adaptive consequences. What happens when we get so angry with an erratic driver that we feel like yelling at him, yet we do not? Or when we feel down but want to be in good spirits for a party? Or when we find something outrageously funny but need to stifle our laughter during a formal ceremony? At times such as these, we

basal levels of GCs permitting other facets of the stress response to emerge efficaciously, and/or by stress levels of GCs actively stimulating the stress response. In con-trast, a revisionist viewpoint posits that GCs suppress the stress response, preventing it from being pathologically overactivated

Abstract not found

Presented here is a genome sequence of an individual human. It was produced from;32 million random DNA fragments, sequenced by Sanger dideoxy technology and assembled into 4,528 scaffolds, comprising 2,810 million bases (Mb) of contiguous sequence with approximately 7.5-fold coverage for any given