propose in this note. 1 Statement of the result Let K be a global field and GK its absolute Galois group. Let A be a finite GK-module. The action of GK on A factors through a finite factor group. Let G be such a factor group, i.e. G = GK/U where U is an open normal subgroup of GK which acts trivially on A

Abstract. Let ∆ be the unit disc of C and let f, g ∈ Hol(∆, ∆) be such that f ◦ g = g ◦ f. For A>1, let FixA(f):={p ∈ ∂ ∆ | limr→1 f(rp) = p, limr→1 |f ′ (rp) | ≤A}. Westudythebehaviorofg on FixA(f). In particular, we prove that g(FixA(f)) ⊆ FixA(f). As a consequence, besides conditions

ABSTRACT. Julia's lemma and Wolff's theorem are established for (Fréchet-) holomorphic maps of bounded symmetric homogeneous domains in infinite dimensional complex Banach spaces called J*-algebras. 1. Introduction. In [11] V. P. Potapov extended the classical lemma of G. Julia [8, p. 87

Abstract. For the isotropic O(3) model we prove that percolation in the Wolff representation is a necessary and sufficient condition for positivity of the spontaneous magnetization. The distinguishing feature of Wolff’s cluster algorithm [W] for models with O(n) symmetry is the apparent fact

Copyright c © 2015 by Anna-Rose G. Wolff

taught us much less about how infants listen to voices than about how they look at faces. Although early investigators suggested that young infants smile earlier to voices than to faces (Wolff, 1963) and respond differen-tially to positive and negative affect in the voice (Buhler & Hetzer, 1928

of the separating matrix. This is the case with most applications (e.g., speech sounds, electroencephalogram sig-nals). It is known that this causes numerical instability in some cases. In order to resolve this difficulty, this article introduces new nonholo-nomic constraints in the learning algorithm

1976). Cells within the furrow arrest in the G1 phase of the cell cycle (Wolff and Ready, 1991). These cells either emerge from the furrow as five-cell preclusters of R2–R5

Objectives.-Migraine is a risk factor for stroke in young women. Biomarker studies implicate endothelial activation as a possible mechanism. Emerging relationships of childhood adversity with migraine, and with inflammation, a component of endothelial activation, suggest that it may play a role in the migraine-stroke association. Our objective is to evaluate the relationship between adverse childhood experiences (ACEs), migraine, and vascular biomarker levels in premenopausal women. Methods.-Vascular and metabolic biomarkers from women 18-50 years, including 125 with migraine (interictal) and 50 without migraine, were evaluated. An ACE questionnaire was later collected by mail (response rate 80.6%, 100 migraineurs, 41 controls). Results.-Migraineurs and controls were demographically similar. Migraineurs reported adversity more commonly than controls (71% vs 46%, odds ratio [OR] = 1.53, 95% confidence interval 1.07-2.17). Average ACE scores were elevated in migraineurs as compared with controls (2.4 vs 0.76, P < .001). ACE scores correlated with headache frequency (0.37, P = .001) and younger age of headache onset (-0.22, P = .04). It also correlated with body mass index (r = 0.43, P = .0001), von Willebrand factor activity (r = 0.21, P = .009), tissue plasminogen activator antigen (r = 0.28, P = .004), prothrombin activation fragment (r = 0.36, P = .001), high-sensitivity C-reactive protein (r = 0.98, P = .0001), transforming growth factor-beta1 (r = 0.28, P = .003), tissue necrosis factor-alpha (r = 0.20, P = .03), interleukin-6 (r = 0.22, P = .03), adiponectin (r = -0.29, P = .003), and nitrate/ nitrite concentration (r = -314, P = .001). Logistic regression analyses (adjusted for vascular risk factors and migraine) demonstrated an association of childhood adversity with inflammatory factors (high-sensitivity C-reactive protein, interleukin-6, and tissue necrosis factor-alpha).

1.What is the psychobiological outcome? (e.g., neuropsychological functioning, immune functioning, telomere erosion). Define and describe it briefly. INFLAMMATION. Inflammation is part of the innate immune response, a powerful defense mechanism that enables recognition of evolutionarily