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The Children’s Hospital
"... these factors in promoting end joining and thereby preventing DSBs from progressing into chromosomal ing to replacement of Cm with a downstream CH gene to complete CSR (Honjo et al., 2004). Notably, g-H2AX ion lly ec-ted p-breaks and translocations. As cellular p53 status does not markedly influence ..."
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these factors in promoting end joining and thereby preventing DSBs from progressing into chromosomal ing to replacement of Cm with a downstream CH gene to complete CSR (Honjo et al., 2004). Notably, g-H2AX ion lly ec-ted p-breaks and translocations. As cellular p53 status does not markedly
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"... ligand family (reviewed by Greenwald, 1994; Artavanis- indicating that this mutant form of Notch has intrinsic signaling tional regulator human C promoter Binding Factor (CBF1), which transactivates the hairy and Enhancer of demonstrate the existence of a CBF1-independent pathway by which Notch inhi ..."
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and biochemical studies have implicated the homologous proteins Drosophila Suppressor of Hairless [Su(H)], C. elegans LAG-1 (Christensen et al., 1996) and mammalian RBP-Jk/CBF1/KBF2 (hereafter referred to as CBF1) in the LIN12/Notch signaling pathway (reviewed by Honjo, 1996; Lewis, 1996). Results from some
