The authors outline a cognitive and computational account of causal learning in children. They propose that children use specialized cognitive systems that allow them to recover an accurate “causal map ” of the world: an abstract, coherent, learned representation of the causal relations among events. This kind of knowledge can be perspicuously understood in terms of the formalism of directed graphical causal models, or Bayes nets. Children’s causal learning and inference may involve computations similar to those for learning causal Bayes nets and for predicting with them. Experimental results suggest that 2to 4-year-old children construct new causal maps and that their learning is consistent with the Bayes net formalism.

Inducing causal relationships from observations is a classic problem in scientific inference, statistics, and machine learning. It is also a central part of human learning, and a task that people perform remarkably well given its notorious difficulties. People can learn causal structure in various settings, from diverse forms of data: observations of the co-occurrence frequencies between causes and effects, interactions between physical objects, or patterns of spatial or temporal coincidence. These different modes of learning are typically thought of as distinct psychological processes and are rarely studied together, but at heart they present the same inductive challenge—identifying the unobservable mechanisms that generate observable relations between variables, objects, or events, given only sparse and limited data. We present a computational-level analysis of this inductive problem and a framework for its solution, which allows us to model all these forms of causal learning in a common language. In this framework, causal induction is the product of domain-general statistical inference guided by domain-specific prior knowledge, in the form of an abstract causal theory. We identify 3 key aspects of abstract prior knowledge—the ontology of entities, properties, and relations that organizes a domain; the plausibility of specific causal relationships; and the functional form of those relationships—and show how they provide the constraints that people need to induce useful causal models from sparse data.

A scheme is described for locally Bayesian parameter updating in models structured as successions of component functions. The essential idea is to back-propagate the target data to interior modules, such that an interior component’s target is the input to the next component that maximizes the probability of the next component’s target. Each layer then does locally Bayesian learning. The approach assumes online trial-by-trial learning. The resulting parameter updating is not globally Bayesian but can better capture human behavior. The approach is implemented for an associative learning model that first maps inputs to attentionally filtered inputs and then maps attentionally filtered inputs to outputs. The Bayesian updating allows the associative model to exhibit retrospective revaluation effects such as backward blocking and unovershadowing, which have been challenging for associative learning models. The back-propagation of target values to attention allows the model to show trial-order effects, including highlighting and differences in magnitude of forward and backward blocking, which have been challenging for Bayesian learning models.

For over 200 years, philosophers and mathematicians have been using probability theory to describe human cognition. While the theory of probabilities was first developed as a means of analyzing games of chance, it quickly took on a larger and deeper significance as a formal account of how rational agents should reason in situations of uncertainty

A pervasive feature of the sciences, particularly the applied sciences, is an experimental focus on a few (often only one) possible causal connections. At the same time, scientists often advance and apply relatively broad models that incorporate many different causal mechanisms. We are naturally led to ask whether there are normative rules for integrating multiple local experimental conclusions into models covering many additional variables. In this paper, we provide a positive answer to this question by developing several inference rules that use local causal models to place constraints on the integrated model, given quite general assumptions. We also demonstrate the practical value of these rules by applying them to a case study from ecology. 1 Experimental scope in applied sciences 2 Fusing the results of experiments 3 A concrete example of the inference rules 4 Application to a case study 1 Experimental scope in applied sciences Total photosynthetic material has increased globally in recent years (though with local decreases), and one might naturally wonder why. In a recent paper in Science, Nemani et al. ([2003]) focused on some of the potential causes of global vegetation growth during the past 20 years. Their analysis focused on only four variables: growing season average temperature, vapor pressure deficit, solar radiation, and net primary production (photosynthetic material). Their study considered only a limited variable set because of (a) the global scale of their analysis, and (b) the relatively long study period (18 years). Despite this limited scope (in terms of variables), their study gives substantial support to the hypothesis that the first three variables are causes of the last, and helps to clarify the functional form of those dependencies. At the same time, they explicitly note that there are many causally relevant variables that were ignored in their study, such as vegetation

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