The primary aim of this paper is to show how graphical models can be used as a mathematical language for integrating statistical and subject-matter information. In particular, the paper develops a principled, nonparametric framework for causal inference, in which diagrams are queried to determine if the assumptions available are sufficient for identifying causal effects from nonexperimental data. If so the diagrams can be queried to produce mathematical expressions for causal effects in terms of observed distributions; otherwise, the diagrams can be queried to suggest additional observations or auxiliary experiments from which the desired inferences can be obtained. Key words: Causal inference, graph models, interventions treatment effect 1 Introduction The tools introduced in this paper are aimed at helping researchers communicate qualitative assumptions about cause-effect relationships, elucidate the ramifications of such assumptions, and derive causal inferences from a combination...

This paper concerns the assessment of the effects of actions or policy interventions from a combination of: (i) nonexperimental data, and (ii) substantive assumptions. The assumptions are encoded in the form of a directed acyclic graph, also called "causal graph", in which some variables are presumed to be unobserved. The paper establishes a necessary and sufficient criterion for the identifiability of the causal effects of a singleton variable on all other variables in the model, and a powerful sufficient criterion for the effects of a singleton variable on any set of variables.

Structural equation modeling (SEM) has dominated causal analysis in the social and behavioral sciences since the 1960s. Currently, many SEM practitioners are having difficulty articulating the causal content of SEM and are seeking foundational answers.

Counterfactual quantities representing path-specific effects arise in cases where we are interested in computing the effect of one variable on another only along certain causal paths in the graph (in other words by excluding a set of edges from consideration). A recent paper [7] details a method by which such an exclusion can be specified formally by fixing the value of the parent node of each excluded edge. In this paper we derive simple, graphical conditions for experimental identifiability of path-specific effects, namely, conditions under which path-specific effects can be estimated consistently from data obtained from controlled experiments. 1

Introduction Reasoning in terms of cause and effect is a strategy that arises in many tasks. For example, diagnosis is usually defined as the task of finding the causes (illnesses) from the observed effects (symptoms). Similarly, prediction can be understood as the description of a future plausible situation where observed effects will be in accordance with the known causal structure of the phenomenon being studied. Causal models are a summary of the knowledge about a phenomenon expressed in terms of causation. Many areas of the ap- # This work has been partially supported by the Spanish Comission Interministerial de Ciencia y Tecnologia Project CICYT-TIC96 -0878. plied sciences (econometry, biomedics, engineering, etc.) have used such a term to refer to models that yield explanations, allow for prediction and facilitate planning and decision making. Causal reasoning can be viewed as inference guided by a causation theory. That kind of inference can be further specialised into induc

INTRODUCTION This chapter surveys the development of graphical models known as Bayesian networks, summarizes their semantical basis and assesses their properties and applications to reasoning and planning. Bayesian networks are directed acyclic graphs (DAGs) in which the nodes represent variables of interest (e.g., the temperature of a device, the gender of a patient, a feature of an object, the occurrence of an event) and the links represent causal influences among the variables. The strength of an influence is represented by conditional probabilities that are attached to each cluster of parents-child nodes in the network. Figure 1 illustrates a simple yet typical Bayesian network. It describes the causal relationships among the season of the year (X 1 ), whether rain falls (X 2 ) during the season, whether the sprinkler is on (X 3 ) during that season, whether the pavement would get wet (X<F28.21

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This paper deals with the problem of estimating the probability of causation, that is, the probability that one event was the real cause of another, in a given scenario. Starting from structural-semantical definitions of the probabilities of necessary or sufficient causation (or both), we show how to bound these quantities from data obtained in experimental and observational studies, under general assumptions concerning the data-generating process. In particular, we strengthen the results of Pearl (1999) by presenting sharp bounds based on combined experimental and nonexperimental data under no process assumptions, as well as under the mild assumptions of exogeneity (no confounding) and monotonicity (no prevention). These results delineate more precisely the basic assumptions that must be made before statistical measures such as the excess-risk-ratio could be used for assessing attributional quantities such as the probability of causation. 1

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This paper aims at assisting empirical researchers benefit from recent advances in causal inference. The paper stresses the paradigmatic shifts that must be undertaken in moving from traditional statistical analysis to causal analysis of multivariate data. Special emphasis is placed on the assumptions that underly all causal inferences, the languages used in formulating those assumptions, and the conditional nature of causal claims inferred from nonexperimental studies. These emphases are illustrated through a brief survey of recent results, including the control of confounding, the assessment of causal effects, the interpretation of counterfactuals, and a symbiosis between counterfactual and graphical methods of analysis.