The primary aim of this paper is to show how graphical models can be used as a mathematical language for integrating statistical and subject-matter information. In particular, the paper develops a principled, nonparametric framework for causal inference, in which diagrams are queried to determine if the assumptions available are sufficient for identifying causal effects from nonexperimental data. If so the diagrams can be queried to produce mathematical expressions for causal effects in terms of observed distributions; otherwise, the diagrams can be queried to suggest additional observations or auxiliary experiments from which the desired inferences can be obtained. Key words: Causal inference, graph models, interventions treatment effect 1 Introduction The tools introduced in this paper are aimed at helping researchers communicate qualitative assumptions about cause-effect relationships, elucidate the ramifications of such assumptions, and derive causal inferences from a combination...

The direct effect of one event on another can be defined and measured by holding constant all intermediate variables between the two. Indirect effects present conceptual and practical difficulties (in nonlinear models), because they cannot be isolated by holding certain variables constant. This paper presents a new way of defining the effect transmitted through a restricted set of paths, without controlling variables on the remaining paths. This permits the assessment of a more natural type of direct and indirect effects, one that is applicable in both linear and nonlinear models and that has broader policy-related interpretations. The paper establishes conditions under which such assessments can be estimated consistently from experimental and nonexperimental data, and thus extends path-analytic techniques to nonlinear and nonparametric models.

Structural equation modeling (SEM) has dominated causal analysis in the social and behavioral sciences since the 1960s. Currently, many SEM practitioners are having difficulty articulating the causal content of SEM and are seeking foundational answers.

This paper studies the causal interpretation of counterfactual sentences using a modifiable structural equation model. It is shown that two properties of counterfactuals, namely, composition and effectiveness, are sound and complete relative to this interpretation, when recursive (i.e., feedback-less) models are considered. Composition and effectiveness also hold in Lewis's closest-world semantics, which implies that for recursive models the causal interpretation imposes no restrictions beyond those embodied in Lewis's framework. A third property, called reversibility, holds in nonrecursive causal models but not in Lewis's closest-world semantics, which implies that Lewis's axioms do not capture some properties of systems with feedback. Causal inferences based on counterfactual analysis are exemplified and compared to those based on graphical models.

This paper summarizes basic concepts and principles that I have found to be useful in dealing with causal reasoning. The paper is written as a companion to a lecture under the same title, to be presented at IJCAI-99, and is intended to supplement the lecture with technical details and pointers to more elaborate discussions in the literature. The ruling conception will be to treat causation as a computational schema devised to identify the invariant relationships in the environment, so as to facilitate reliable prediction of the effect of actions. This conception, as well as several of its satellite principles and tools, has been guiding paradigm for several research communities in AI, most notably those connected with causal discovery, troubleshooting, planning under uncertainty and modeling the behavior of physical systems. My hopes are to encourage a broader and more effective usage of causal modeling by explicating these common principles in simple and familiar mathematical form. Af...

Chain graphs are a natural generalization of directed acyclic graphs (DAGs) and undirected graphs. However, the apparent simplicity of chain graphs belies the subtlety of the conditional independence hypotheses that they represent. There are a number of simple and apparently plausible, but ultimately fallacious interpretations of chain graphs that are often invoked, implicitly or explicitly. These interpretations also lead to awed methods for applying background knowledge to model selection. We present a valid interpretation by showing how the distribution corresponding to a chain graph may be generated as the equilibrium distribution of dynamic models with feedback. These dynamic interpretations lead to a simple theory of intervention, extending the theory developed for DAGs. Finally, we contrast chain graph models under this interpretation with simultaneous equation models which have traditionally been used to model feedback in econometrics. Keywords: Causal model; cha...

INTRODUCTION This chapter surveys the development of graphical models known as Bayesian networks, summarizes their semantical basis and assesses their properties and applications to reasoning and planning. Bayesian networks are directed acyclic graphs (DAGs) in which the nodes represent variables of interest (e.g., the temperature of a device, the gender of a patient, a feature of an object, the occurrence of an event) and the links represent causal influences among the variables. The strength of an influence is represented by conditional probabilities that are attached to each cluster of parents-child nodes in the network. Figure 1 illustrates a simple yet typical Bayesian network. It describes the causal relationships among the season of the year (X 1 ), whether rain falls (X 2 ) during the season, whether the sprinkler is on (X 3 ) during that season, whether the pavement would get wet (X<F28.21

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This paper deals with the problem of estimating the probability of causation, that is, the probability that one event was the real cause of another, in a given scenario. Starting from structural-semantical definitions of the probabilities of necessary or sufficient causation (or both), we show how to bound these quantities from data obtained in experimental and observational studies, under general assumptions concerning the data-generating process. In particular, we strengthen the results of Pearl (1999) by presenting sharp bounds based on combined experimental and nonexperimental data under no process assumptions, as well as under the mild assumptions of exogeneity (no confounding) and monotonicity (no prevention). These results delineate more precisely the basic assumptions that must be made before statistical measures such as the excess-risk-ratio could be used for assessing attributional quantities such as the probability of causation. 1

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